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Asthma

SNOMED: 340150071003 wordsUpdated 03/03/2026
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Exam Tips

  • In OSCEs, emphasise variability: variable symptoms + variable expiratory airflow limitation is central to diagnosis.
  • Uncontrolled asthma includes exacerbations needing oral steroids and frequent reliever use (e. g, >=3 days/week) or night waking.
  • Always mention inhaler technique and adherence before stepping up treatment; this is a common exam trap.
  • State explicitly that LABA monotherapy is unsafe in asthma; LABA should be paired with ICS.
  • Red flags for asthma death risk: previous ICU/intubation, recent admission, SABA overuse, poor ICS adherence, psychosocial problems, food allergy/anaphylaxis.
  • Differentiate difficult-to-treat asthma (modifiable factors/comorbidity/adherence) from true severe refractory asthma.

Definition

Asthma is a chronic inflammatory airway disorder with variable expiratory airflow limitation and bronchial hyper-responsiveness, causing recurrent wheeze, breathlessness, chest tightness, and cough. Symptoms fluctuate over time (often worse at night/early morning) and may be spontaneous or trigger-related, with acute exacerbations ranging from mild to life-threatening.

Pathophysiology

Core mechanisms are type 2 and non-type 2 airway inflammation, episodic bronchoconstriction, mucosal oedema, and mucus hypersecretion. In allergic disease, allergen exposure drives Th2 cytokines (IL-4, IL-5, IL-13), IgE production, eosinophilic inflammation, and mast-cell mediator release; viral infection, pollutants, and irritants can amplify this. Repeated inflammation leads to airway remodelling (subepithelial fibrosis, smooth-muscle hypertrophy, goblet-cell hyperplasia), explaining persistent symptoms and possible fixed airflow limitation in some patients. See Figure: schematic of airway inflammation and remodelling in standard respiratory pathology texts.

Risk Factors

  • Personal or family atopy (asthma, eczema, allergic rhinitis, nasal polyps)
  • Allergen sensitization (house dust mite, animal dander, pollens, mould)
  • Comorbid allergic rhinitis/chronic rhinosinusitis (especially with nasal polyps)
  • Food allergy (especially in children)
  • Smoking exposure (active/passive; including vaping aerosols)
  • Obesity and deconditioning
  • Preterm birth or low birth weight
  • Occupational exposure (e. g, flour dust, isocyanates, cleaning chemicals, laboratory animals, wood dust, welding fumes)
  • Female sex for persistent adult asthma; male predominance in childhood
  • Drugs that can worsen asthma (non-selective beta-blockers, aspirin/NSAIDs in sensitive patients)

Clinical Features

Symptoms

  • Episodic wheeze
  • Shortness of breath
  • Chest tightness
  • Cough (often nocturnal or early morning; may be cough-variant asthma)
  • Variable symptoms triggered by exercise, viral URTI, allergens, cold air, laughter/crying, smoke, weather changes
  • Nocturnal waking and reliever overuse in poor control

Signs

  • Expiratory polyphonic wheeze
  • Prolonged expiratory phase
  • Tachypnoea and tachycardia during exacerbation
  • Use of accessory respiratory muscles in acute attacks
  • Reduced peak expiratory flow versus personal best/predicted
  • Severe/life-threatening features: inability to complete sentences, exhaustion, silent chest, cyanosis, altered consciousness

Investigations

Spirometry with bronchodilator reversibility:Obstructive pattern with variable airflow limitation; post-bronchodilator FEV1 increase typically >=12% and >=200 mL supports asthma
Peak expiratory flow (PEF) monitoring (2-4 weeks):Diurnal variability >20% supports asthma
Fractional exhaled nitric oxide (FeNO):Raised FeNO supports eosinophilic/type 2 airway inflammation (commonly >=50 ppb in adults, >=35 ppb in children 5-16 years)
Blood eosinophil count:Raised eosinophils (e. g, >=0.3 x 10^9/L) supports type 2 inflammation and may guide biologic eligibility
Allergy testing (specific IgE/skin-prick):Identifies sensitization and relevant trigger profile
Chest X-ray:Often normal in stable asthma; mainly to exclude alternative diagnoses or complications (pneumothorax, pneumonia)
Bronchial challenge testing (if diagnosis remains uncertain):Airway hyper-responsiveness supports diagnosis

Management

Lifestyle Modifications

  • Provide a personalised written asthma action plan with clear escalation and emergency advice
  • Check and correct inhaler technique at every review; reinforce adherence
  • Smoking cessation and avoidance of passive smoke/vape exposure
  • Trigger reduction: allergen/occupational exposure control; consider workplace assessment for suspected occupational asthma
  • Weight optimisation, physical activity, and management of rhinitis/sinus disease/reflux/anxiety where relevant
  • Annual influenza vaccination; pneumococcal vaccination as clinically indicated

Pharmacological Treatment

Reliever (SABA) for breakthrough symptoms

  • Salbutamol inhaler 100 micrograms/puff: 1-2 puffs when required (typical max 200 micrograms up to 4 times daily in routine use)
  • Terbutaline dry powder inhaler 500 micrograms per inhalation when required

Frequent SABA need indicates poor control and increased risk; review urgently if reliever needed >=3 days/week. In acute attacks, repeated high-dose SABA via spacer/nebuliser is used per emergency protocol.

Inhaled corticosteroid (ICS) maintenance

  • Beclometasone dipropionate (standard particle) 100-200 micrograms twice daily (low-dose range)
  • Budesonide 200-400 micrograms twice daily
  • Fluticasone propionate 100-250 micrograms twice daily

ICS is foundational controller therapy. Advise mouth rinsing after use to reduce candidiasis/dysphonia. Monitor growth in children and adrenal effects at prolonged high dose.

Combination ICS/LABA (including MART where appropriate)

  • Budesonide/formoterol DPI 200/6: typically 1 inhalation twice daily maintenance, plus 1 inhalation as needed for symptoms (MART); usual maximum 12 inhalations/day total
  • Beclometasone/formoterol 100/6: maintenance dosing with additional reliever use in MART regimens according to product limits

Do not use LABA without ICS in asthma. MART reduces severe exacerbations in suitable patients; ensure patient understands maximum daily inhalations and when to seek urgent care.

Add-on controller options

  • Montelukast 10 mg at night (adults)
  • Tiotropium Respimat 5 micrograms once daily
  • Modified-release theophylline 200-400 mg twice daily (titrate to plasma levels)

Montelukast: warn about possible neuropsychiatric adverse effects (sleep disturbance, mood/behaviour change). Theophylline has a narrow therapeutic index and major interactions (e. g, macrolides, quinolones); monitor levels and toxicity.

Acute exacerbation treatment

  • Prednisolone 40-50 mg orally once daily for at least 5 days (or until recovery)
  • Nebulised salbutamol 2.5-5 mg repeated as needed; add nebulised ipratropium bromide 500 micrograms in severe attacks
  • Intravenous magnesium sulfate 1.2-2 g over 20 minutes for severe/life-threatening exacerbation not responding adequately

Assess severity early and escalate promptly if life-threatening features. Avoid sedation. Give controlled oxygen to target saturation 94-98% unless risk of hypercapnic failure dictates otherwise.

Severe eosinophilic/allergic asthma biologics (specialist care)

  • Mepolizumab 100 mg subcutaneously every 4 weeks
  • Benralizumab 30 mg subcutaneously every 4 weeks for first 3 doses, then every 8 weeks
  • Dupilumab 200 mg or 300 mg subcutaneously every 2 weeks (indication-dependent)
  • Tezepelumab 210 mg subcutaneously every 4 weeks
  • Omalizumab subcutaneous dose based on IgE and body weight

Reserved for uncontrolled severe asthma after optimisation of standard therapy. Monitor for hypersensitivity/anaphylaxis and reassess response regularly.

Complications

  • Acute severe or life-threatening exacerbation with respiratory failure
  • Pneumothorax or pneumomediastinum during severe attacks
  • Secondary pneumonia
  • Persistent airflow limitation from airway remodelling (fixed obstruction phenotype)
  • Reduced quality of life, school/work absence, and exercise limitation
  • Anxiety/depression with poorer adherence and control
  • Asthma-related mortality (higher risk with prior near-fatal attack, recent hospitalisation, poor ICS adherence, or SABA overuse)
  • Occupational consequences including job loss and progressive decline if exposure continues

Prognosis

Course is variable: some children remit, but many have persistent or relapsing disease into adulthood, especially with severe childhood symptoms, atopy, smoke exposure, or airway hyper-responsiveness. Adult-onset asthma is less likely to remit, and severe disease may need long-term high-intensity treatment. Prognosis improves with early diagnosis, consistent ICS-based therapy, trigger control (including occupational exposure cessation), and robust self-management planning.

Sources & References

🏥BMJ Best Practice(1)

💊BNF Drug References(50)

NICE Guidelines(1)

📖Textbook References(1)

  • David Randall PhD MRCP (Editor), John Booth PhD MRCP (Editor), K - Kumar and Clark's Clinical Medicine (2025, American Elsevier Publishing Co.) - libgen.li.pdf(pp. 581)[context]

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