6 quiz questions available for this topicTake Quiz

Hand, foot, and mouth disease

SNOMED: 54222004691 wordsUpdated 03/03/2026
💡

Exam Tips

  • Classic OSCE triad: mild fever + painful oral ulcers + acral papulovesicular rash (hands/feet, often dorsolateral digits).
  • A6 clue: wider vesiculobullous eruption, perioral involvement, flexural/eczema-site accentuation, later peeling/nail shedding.
  • Investigations are usually unnecessary in primary care; reserve PCR typing for severe/atypical, outbreak, pregnancy/neonatal, or EV71-risk travel contexts.
  • Always assess hydration status and give explicit red-flag safety-netting; this is frequently examined.
  • Do not prescribe antibiotics routinely; avoid aspirin in under-16s and use ibuprofen cautiously if dehydrated.

Definition

Hand, foot and mouth disease (HFMD) is an acute, usually self-limiting enteroviral infection (most often Coxsackie A16 or A6) that causes painful oral enanthem with a papulovesicular exanthem on distal limbs. It typically affects children under 10 years, has an incubation of about 3-5 days, and must be distinguished from animal foot-and-mouth disease, which is caused by a different virus.

Pathophysiology

After exposure (respiratory secretions, blister fluid, or faeco-oral spread), enteroviruses replicate in pharyngeal and intestinal lymphoid tissue, then disseminate via lymphatics/blood to skin and oral mucosa, producing vesicles that rapidly ulcerate in the mouth. Picornaviruses are non-enveloped single-stranded RNA viruses with environmental resilience, which helps transmission in childcare settings. Serotype influences phenotype: Coxsackie A6 is associated with more extensive vesiculobullous rash, higher fever, later desquamation/onychomadesis, and more adult infection; EV71 (rare in Europe) has greater neurotropism and risk of brainstem encephalitis/autonomic dysregulation.

Risk Factors

  • Age under 10 years (especially under 4-5 years)
  • Nursery/school attendance or household contact with infected child
  • Poor hand hygiene, overcrowding, limited clean water access, large household size
  • Immunocompromise or immunosuppressive therapy
  • Recent travel to East or Southeast Asia (raises concern for EV71)
  • Maternal peripartum infection (risk of neonatal transmission)

Clinical Features

Symptoms

  • Prodrome (12-36 hours): low-grade fever (often 38-39 C), malaise, reduced appetite, sore throat/mouth
  • Oral pain causing reduced intake or dehydration risk
  • Myalgia, cough, abdominal pain; occasional vomiting (more reported with EV71)
  • Rash discomfort or pain on hands/feet; sometimes asymptomatic skin lesions
  • In atypical A6 disease: higher fever and more widespread rash, later skin peeling

Signs

  • Oral lesions: 2-8 mm erythematous macules/papules progressing to vesicles then shallow yellow-grey ulcers with erythematous halo (hard palate, tongue, buccal mucosa, lips/pharynx)
  • Skin lesions: 2-5 mm erythematous macules/papules with central grey vesicle, often elliptical and aligned with skin lines
  • Distribution: sides/dorsum of fingers and hands, margins of heels/feet, sometimes buttocks/groin (especially infants)
  • Atypical A6 pattern: more diffuse eruption including eczema-prone flexures, possible petechiae/purpura or larger bullae; prominent perioral cutaneous lesions
  • Possible strawberry tongue in adults; erosive napkin dermatitis in young children

Investigations

Clinical diagnosis (history + examination):Typical oral ulcers plus acral papulovesicular rash in a mild febrile viral illness
Hydration assessment (clinical):Look for reduced oral intake, dry mucosa, low urine output, tachycardia or delayed capillary refill
Enterovirus PCR from throat/stool/vesicle swab (selective use):Confirms enterovirus/serotype in atypical, severe, outbreak, late-pregnancy, neonatal, or travel-associated suspected EV71 cases

Management

Lifestyle Modifications

  • Encourage regular fluids and soft/cool foods; monitor urine output to prevent dehydration
  • Hand hygiene, cleaning shared surfaces, and avoiding sharing cups/cutlery/towels
  • Child can usually return to nursery/school when clinically well enough; prolonged exclusion is not required solely for rash because viral shedding may persist
  • Give clear safety-net advice: urgent review for poor intake, drowsiness, persistent high fever, severe headache, neck/back stiffness, breathing difficulty, or reduced responsiveness
  • See DermNet clinical image sets for pattern recognition of typical and atypical lesions

Pharmacological Treatment

Analgesic/antipyretic

  • Paracetamol oral: child 3 months-11 years 15 mg/kg every 4-6 hours as needed (max 4 doses in 24 hours); age 12-17 years 500 mg-1 g every 4-6 hours (max 4 g/day)

First-line for fever and mouth pain; dose by weight in children; avoid unintentional overdose from combination products.

NSAID (if needed, second-line)

  • Ibuprofen oral: child 3 months-11 years 5-10 mg/kg per dose 3-4 times daily (usual max 30 mg/kg/day); age 12-17 years 200-400 mg up to 3 times daily

Avoid/caution in dehydration, renal impairment, active GI ulceration, NSAID hypersensitivity/asthma sensitivity, and chickenpox due to risk of severe skin/soft tissue complications.

Complications

  • Dehydration from painful oral lesions (most common)
  • Secondary bacterial infection of skin lesions (uncommon)
  • Post-infectious nail changes (Beau lines, leukonychia, discolouration, onychomadesis) weeks after illness
  • Rare severe neurological/cardiopulmonary disease with EV71 (brainstem encephalitis, aseptic meningitis, acute flaccid paralysis, autonomic instability, pulmonary oedema, myocardial dysfunction)
  • Pregnancy/perinatal concerns: possible neonatal infection if maternal infection occurs around delivery; severe neonatal systemic disease is rare

Prognosis

Overall prognosis is excellent: most children recover fully within 7-10 days without specific antiviral treatment. Recurrence can occur because immunity is serotype-specific (limited cross-protection between enteroviruses). A6 infections may be followed by temporary desquamation and nail shedding; severe long-term sequelae are uncommon and mainly linked to rare EV71-associated severe disease.

Sources & References

NICE Guidelines(1)

📖Textbook References(2)

  • David Randall PhD MRCP (Editor), John Booth PhD MRCP (Editor), K - Kumar and Clark's Clinical Medicine (2025, American Elsevier Publishing Co.) - libgen.li.pdf(pp. 1496)[context]
  • Oxford Handbook of Clinical Diagnosis (Huw Llewelyn, Hock Aun Ang, Keir Lewis etc.) (Z-Library).pdf(pp. 156)[context]

Test Your Knowledge

6 quiz questions available for this topic

Start Quiz