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Olecranon bursitis

SNOMED: 156373110001191051005 wordsUpdated 03/03/2026
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Exam Tips

  • High-yield distinction: bursitis gives a focal fluctuant posterior-elbow swelling with near-normal passive/active elbow range, whereas septic arthritis causes painful global restriction.
  • Do not rely on fever alone to exclude sepsis; many septic cases are afebrile, and warmth/tenderness overlap with non-septic bursitis.
  • In OSCEs, state clearly that aspiration should be performed using sterile technique and sent for Gram stain, culture, and crystals before antibiotics when safe.
  • Always mention risk factors (skin break, immunosuppression, diabetes, prior aspiration, gout/RA) and red flags (tachycardia, hypotension, confusion).
  • Include a safety warning: avoid steroid injection when infection is possible; untreated septic bursitis can progress to bacteraemia or osteomyelitis.

Definition

Olecranon bursitis is inflammation of the superficial synovial bursa over the olecranon process, producing a localized, fluctuant posterior-elbow swelling. It is usually non-septic (often after repetitive minor trauma or a direct blow), but may be septic when organisms seed the bursa through broken skin; importantly, the bursa does not normally communicate with the elbow joint.

Pathophysiology

Mechanical irritation or trauma causes bursal microvascular injury, haemorrhage, and release of inflammatory mediators, leading to synovial hyperplasia, thickening of the bursal wall, increased capillary permeability, and protein-rich fluid accumulation. In septic disease, organisms (most commonly Staphylococcus aureus, then streptococci) enter through skin breaches or iatrogenic puncture, triggering neutrophil-predominant inflammation and purulent/turbid fluid. Crystal disease (especially gout) can inflame the bursa via monosodium urate deposition, and chronic inflammation may lead to recurrent effusions, fibrosis, sinus formation, or (rarely) extension to adjacent bone/joint in high-risk patients.

Risk Factors

  • Repeated elbow pressure (leaning on elbows) or occupational microtrauma (for example mechanics, plumbers, roofers, carpet layers, truck drivers, students/writers)
  • Single direct trauma to the posterior elbow
  • Sports with repetitive elbow loading or impact (for example rugby, hockey, gymnastics, weightlifting, throwing sports)
  • Gout and rheumatoid arthritis (including rheumatoid nodules)
  • Immunocompromise (diabetes, HIV, alcohol misuse, systemic corticosteroid therapy)
  • Skin abrasion/laceration over the olecranon
  • Previous bursal aspiration/injection or pre-existing bursal disease
  • Male sex, young to middle age
  • Less common: haemodialysis positioning, chronic lung disease with prolonged elbow bracing

Clinical Features

Symptoms

  • Swelling over the tip of the elbow developing over hours to days
  • Pain or tenderness (may be minimal in non-septic cases)
  • Warmth and discomfort when leaning on the elbow
  • Pain at terminal flexion from compression of the swollen bursa
  • Systemic upset in severe septic cases (fever, malaise)

Signs

  • Localized, fluctuant, compressible swelling over the olecranon process
  • Elbow joint movement usually preserved, except pain at full flexion
  • Erythema and increasing tenderness suggest possible sepsis
  • Overlying cellulitis, abrasion, or puncture wound supports septic cause
  • Red flags for severe infection: tachycardia, hypotension, altered mental state
  • See Figure: classic posterior elbow 'golf-ball' fluctuant swelling over olecranon in clinical examination atlases

Investigations

Clinical assessment (history + focused elbow exam):Localized bursal swelling with relatively preserved joint movement favors olecranon bursitis over septic arthritis
Bursal aspiration (sterile technique, ideally before antibiotics if sepsis suspected):Fluid may be purulent/turbid (septic likely), straw-colored (often non-septic), blood-stained (trauma/sepsis/crystal/inflammatory), or milky (crystal arthropathy)
Bursal fluid Gram stain and culture:Identifies causative organism (commonly S. aureus or streptococci) and guides targeted antibiotics
Bursal fluid crystal analysis (polarized microscopy):Monosodium urate crystals suggest gout; calcium pyrophosphate suggests pseudogout
Bursal fluid white cell differential and glucose:High neutrophil count and low glucose support infection when Gram/culture are initially negative
Blood tests (FBC, CRP/ESR, U&Es, glucose; serum urate if relevant):Inflammatory markers may rise in septic or inflammatory causes; urate can support gout context but is not diagnostic acutely
Blood cultures (if febrile/systemically unwell):May detect bacteraemia in severe septic bursitis
Elbow X-ray (if trauma, very rapid swelling, or bony concern):May show fracture, olecranon spur, or other bony pathology rather than isolated bursitis
Ultrasound (if diagnostic uncertainty):Confirms superficial fluid collection and helps distinguish bursal swelling from soft-tissue mass

Management

Lifestyle Modifications

  • Relative rest; avoid leaning/pressure on the elbow and modify precipitating activities/work tasks
  • Elbow padding/protective sleeve and ergonomic adjustments to reduce recurrence
  • Ice packs 10-15 minutes several times daily in acute non-septic inflammation
  • Compression bandage if comfortable and no neurovascular compromise
  • Safety-net advice: seek urgent review for fever, spreading erythema, worsening pain, or systemic symptoms

Pharmacological Treatment

Analgesia/anti-inflammatory for non-septic bursitis

  • Paracetamol 1 g orally every 4-6 hours as needed (max 4 g/day)
  • Ibuprofen 400 mg orally three times daily with food (typical max 1.2 g/day OTC; up to 2.4 g/day on prescription if appropriate)
  • Naproxen 500 mg orally twice daily (with gastroprotection when indicated)

Avoid/limit NSAIDs in CKD, heart failure, active peptic ulcer disease, or high GI/CV risk; consider a PPI such as omeprazole 20 mg once daily in higher GI-risk adults.

Empirical antibiotics for suspected septic olecranon bursitis (then tailor to culture)

  • Flucloxacillin 500 mg to 1 g orally four times daily for 7 days (extend to 10-14 days by clinical response)
  • If severe/systemically unwell: flucloxacillin 1-2 g IV every 6 hours
  • Penicillin allergy alternative: clarithromycin 500 mg orally twice daily
  • Alternative when macrolide unsuitable: doxycycline 200 mg on day 1, then 100 mg once daily

Take aspirate before first dose when feasible. Follow local antimicrobial policy and sensitivities. Escalate urgently if sepsis features, immunocompromise, or treatment failure.

Crystal-associated bursitis treatment (when gout confirmed/likely)

  • Colchicine 500 micrograms orally two to four times daily until pain improves (max 6 mg per course)
  • Naproxen 500 mg orally twice daily as an alternative if not contraindicated

Reduce colchicine dose in renal impairment and avoid strong CYP3A4/P-gp inhibitors; do not use repeated high-dose courses due to toxicity risk.

Surgical / Interventional

  • Needle aspiration for diagnostic confirmation and pressure relief when sepsis is suspected or diagnosis is uncertain
  • Repeat aspiration may be used in selected persistent septic cases with close follow-up
  • Incision and drainage for loculated/persistent infection or failure of aspiration plus antibiotics
  • Bursectomy for recurrent, refractory, or chronically thickened bursitis after conservative management
  • Do not inject corticosteroid into a bursa if infection is suspected (risk of worsening sepsis)

Complications

  • Recurrence or chronic bursitis (septic or non-septic), especially with ongoing elbow trauma
  • Sinus tract or fistula formation (spontaneous rupture or post-procedural)
  • Secondary infection after aspiration
  • Cellulitis extension
  • Rare secondary septic arthritis (higher risk in chronic inflammatory arthropathy such as rheumatoid arthritis)
  • Bacteraemia, sepsis, or toxic shock syndrome in severe infection
  • Olecranon osteomyelitis from persistent infection
  • Necrotizing soft-tissue infection (very rare)

Prognosis

Overall prognosis is good: most cases settle without major sequelae, and non-septic bursitis is usually benign with conservative treatment. Recurrence is common if repetitive pressure continues; aspiration alone may leave persistent effusion in a minority (historically around 25% at 8 weeks, about 10% at 6 months). Septic bursitis usually resolves with timely aspiration and appropriate antibiotics, although complete clinical resolution can take several weeks.

Sources & References

🏥BMJ Best Practice(1)

NICE Guidelines(1)

📖Textbook References(10)

  • Netter F. Netter Atlas of Human Anatomy. A Systems Approach 8ed 2022.pdf(pp. 2249, 2250)[context]
  • Netter F. Netter Atlas of Human Anatomy. A Systems Approach 8ed 2022.pdf(pp. 2250, 2251)[context]
  • Netter F. Netter Atlas of Human Anatomy. A Systems Approach 8ed 2022.pdf(pp. 2250, 2251)[context]
  • _OceanofPDF.com_Netters_Anatomy_-_8th_edition_-_Frank_H_Netter_MD.pdf(pp. 2249, 2250)[context]
  • _OceanofPDF.com_Netters_Anatomy_-_8th_edition_-_Frank_H_Netter_MD.pdf(pp. 2250, 2251)[context]
  • _OceanofPDF.com_Netters_Anatomy_-_8th_edition_-_Frank_H_Netter_MD.pdf(pp. 2250, 2251)[context]
  • [Williams, Bailey and Love's Short Practice of Surgery] Norman Williams, Christopher Bulstrode, P Ronan O'Connell - Bailey & Love's Short Practice of Surgery 26E (2013, CRC Press) - libgen.li.pdf(pp. 513)[context]
  • [Williams, Bailey and Love's Short Practice of Surgery] Norman Williams, Christopher Bulstrode, P Ronan O'Connell - Bailey & Love's Short Practice of Surgery 26E (2013, CRC Press) - libgen.li.pdf(pp. 513, 514)[context]
  • [Williams, Bailey and Love's Short Practice of Surgery] Norman Williams, Christopher Bulstrode, P Ronan O'Connell - Bailey & Love's Short Practice of Surgery 26E (2013, CRC Press) - libgen.li.pdf(pp. 512, 513)[context]
  • [Williams, Bailey and Love's Short Practice of Surgery] Norman Williams, Christopher Bulstrode, P Ronan O'Connell - Bailey & Love's Short Practice of Surgery 26E (2013, CRC Press) - libgen.li.pdf(pp. 514, 515)[context]

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